Laboratory evidence ranges from increased abnormal ABR interwave intervals I–III and I–V and decreased amplitude waves III and V to absent ABRs, and MRI shows acute abnormalities in the globus pallidus and subthalamic nucleus.
Abnormal ABRs may improve or normalize with exchange transfusion
Since hyperbilirubinemia affects the auditory brainstem and perhaps the auditory nerve, OAEs along with CM will be normal in children deaf due to hyperbilirubinemia and kernicterus. OAE hearing screening alone will miss AN.

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Results: Four neonates showed abnormal or no response and the other seven demonstrated normal response in ABR. All 11 neonates passed TEOAEs. Two neonates showed improvement in auditory function at 3- or 6-month follow-up ABR.
Conclusion: The results of this study indicate that the site of lesion in hearing loss caused by hyperbilirubinemia may be at the retrocochlear location while the cochlea remains intact. TEOAEs may have limitations in evaluation of hearing in the neonates with hyperbilirubinemia.